myelopathy
Thorakale
Myelopathie nach Bestrahlung bei ossär metastasiertem Mammakarzinom. Links T1, rechts T2 sagittal: man erkennt die fettige Degeneration des knöchernen Markraums der bestrahlten Wirbel und besser in T2 als in T1 die zentrale, spindelförmige Myelopathie im unteren Bereich des Bestrahlungsfeldes.
Radiological
approach to non-compressive myelopathies. Clinico-radiological approach to myelopathy
Location,
length, and enhancement: systematic approach to differentiating intramedullary spinal cord lesions. Differentiating intramedullary pathology: location within the cord
Location,
length, and enhancement: systematic approach to differentiating intramedullary spinal cord lesions. Intramedullary spinal cord anatomy
Location,
length, and enhancement: systematic approach to differentiating intramedullary spinal cord lesions. Differentiating intramedullary pathology: short segment (left) versus long segment (right) lesions
Long-term
course of anterior spinal cord herniation presenting with an upper motor neuron syndrome: case report illustrating diagnostic and therapeutic implications. Longitudinal course of anterior spinal cord herniation. a Magnifications of the Th2/Th3 segment obtained in T2-weighted sagittal imaging at different visits. Arrowheads highlight evolving anterior spinal cord herniation and a postoperative myelopathy signal. The red arrow highlights the early subtle sign of ventral spinal cord misplacement and posterior indentation. b Corresponding axial images of Th2/3. c MEP including transcranial (lines 1–2) and lumbar stimulation (lines 3–4) to the right (R) and to the left (L) tibialis anterior muscle. Central motor conductance was pathological after 8 years follow-up (arrow; amplitude R 1.27 mV, L 2.88 mV). After 11 years, spasticity had progressed to the left leg, along with affection of the left leg central motor conductance (arrow; R 1.0 mV, L 2.47 mV). Six months after neurosurgery, there were no significant changes in MEP (R 0.68 mV, L 3.02 mV). Five years after neurosurgery, cortical MEP were absent to the right leg (arrow; R –, L 2.59 mV), despite unchanged MRI. d SEP-recordings during follow-up visits, obtained from stimulation of the right (R) and left (L) tibial nerve. At 8 years follow-up, SEP P40-latency was prolonged bilaterally (R 51.8 ms, L 52.0 ms). At presentation with Brown-Séquard syndrome involving right-sided hypesthesia, SEP from the right was absent (arrow; R –, L 50.8 ms). Six months after neurosurgery, SEP from the right gradually improved (arrow; R 51.2 ms, L 51.2 ms). Five years after neurosurgery, SEP from the right leg was absent again (arrow; R –, L 53.6 ms), despite unchanged MRI. Scale: C, 20 ms/ Div. and 2000 μV/ Div.; D, 20 ms/ Div. and 1.0 μV/ Div. f/u follow-up, n/a not available
Assoziationen und Differentialdiagnosen zu Myelopathie:
