Knochenmarködem-Syndrom des Knies

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course of intra-articular shifting bone marrow edema syndrome of the knee. Shift of bone marrow edema.

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course of intra-articular shifting bone marrow edema syndrome of the knee. 48-year-old man with a large BME in the medial femoral condyle not limited to the subchondral bone on the initial T2-weighted MR investigation (STIR).

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course of intra-articular shifting bone marrow edema syndrome of the knee. The MR investigation shows a shift of a large BME to the medial and the lateral tibial plateau, and a reduction in the size of the BME in the medial femoral condyle 3 months later.

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course of intra-articular shifting bone marrow edema syndrome of the knee. Complete normalization of the MRI signal pattern after 6 months of conservative treatment.

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course of intra-articular shifting bone marrow edema syndrome of the knee. 44-year-old woman with a extensive BME in the medial femoral condyle which is not limited to the subchondral area on the T2-weighted MR investigation (STIR).

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course of intra-articular shifting bone marrow edema syndrome of the knee. The MR image shows a shift of the large BME to the lateral femoral condyle, reduction in the size of the BME in the medial femoral condyle, and remission at the medial tibial condyle 4 months later.

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course of intra-articular shifting bone marrow edema syndrome of the knee. Complete normalization of the MR imaging signal pattern after 10 months.

Extracorporeal

shock wave therapy in the treatment of primary bone marrow edema syndrome of the knee: a prospective randomised controlled study. Pre- (a) and 6 months posttreatment (b) T2-weighted images showing the normalization of a large bone marrow edema within the medial femoral condyle of the left knee, in a 62-year old male patient. (Note: The patient consented to publish the specific information.)

Subchondral

insufficiency fractures, subchondral insufficiency fractures with osteonecrosis, and other apparently spontaneous subchondral bone lesions of the knee—pathogenesis and diagnosis at imaging. Other features associated with bone-marrow-like signal. In case of intense BME-like signal intensity, the area immediately adjacent to the subchondral bone plate may show a very intense, finely linear high signal on fat-suppressed T2-weighted images (arrowheads), which could be linked to hyperemia of this richly vascularized area. Edema-like high signal intensity may also involve the neighboring soft tissues (arrow)

Subchondral

insufficiency fractures, subchondral insufficiency fractures with osteonecrosis, and other apparently spontaneous subchondral bone lesions of the knee—pathogenesis and diagnosis at imaging. Borderline thickness of bone marrow low signal on T2-weighted image. a, b MRI shows low signal intensity on T1-weighted (a) and high signal intensity on fat-suppressed T2-weighted images in the medial femoral condyle (b) (asterisks). Immediately near the subchondral surface, fat-suppressed T2-weighted image shows a very thin layer of tissue of borderline thickness with low signal intensity (arrow in b). c, d In the present case, the follow-up at 3 months showed healing, with normalization of the signal in the inferior pole of the condyle

Subchondral

insufficiency fractures, subchondral insufficiency fractures with osteonecrosis, and other apparently spontaneous subchondral bone lesions of the knee—pathogenesis and diagnosis at imaging. Diagram summarizing the prognostic value of typical subchondral signal intensity on T2-weighted sequences in SIF and SIF-ON. Note that in some cases, a very limited area with subchondral low T2 signal intensity is of doubtful prognosis and may progress either to healing or to SIF-ON. Without a particular event (meniscectomy or rapid cartilage degeneration for example), the evolution of a typical SIF towards a SIF-ON is very rare