Hill-Sachs lesion
A Hill-Sachs defect is a posterolateral humeral head depression fracture, resulting from the impaction with the anterior glenoid rim, therefore indicative of an anterior glenohumeral dislocation. It is often associated with a Bankart lesion of the glenoid.
Terminology
A Hill-Sachs defect is the terminology of preferenceover other terms, such as Hill-Sachs lesions, and Hill-Sachs fractures . Repeat dislocations lead to larger defects, which can result in an "engaging" Hill-Sachs defect, which engages the anterior glenoid when the shoulder is abducted and externally rotated . (see article: On-track and off-track shoulder lesions for further discussion) .
Pathology
Anterior glenohumeral dislocation will lead to impaction of the posterolateral humeral head and anterior glenoid rim. Repeat dislocations can lead to further bony defects in both the humeral head and glenoid and the engaging Hill–Sachs defect is associated with decreased glenoid bone stock, glenoid rim fracture, and chronic instability . Bankart lesions are up to 11 times more common in patients with a Hill-Sachs lesion, with increasing incidence with increasing size .
Radiographic features
When a Hill-Sachs defect is identified careful assessment of the anterior glenoid should be undertaken to assess for a Bankart lesion.
Plain radiograph
- wedge shape defect in the posterolateral aspect of the humeral head
- best appreciated on AP internal rotation view
- smaller defects can be difficult to identify
- on abduction-internal rotation views, the physiological depression at humeral head-neck junction should not be mistaken from Hill-Sachs defect and is evident 2cm from superior humeral head margin
CT and MRI
- loss of the normal circular shape in the posterolateral region of the superior humeral head on axial images
- MRI and CT will show smaller defects
- anatomic shape can be preserved but the presence of bone marrow edema in the posterolateral humeral head indicates an acute injury
- normal flattening of the posterolateral humeral head caudal to the level of coracoid should not be misinterpreted as a Hill-Sachs lesion (sometimes termed pseudo-Hill-Sachs defect)
Treatment and prognosis
The bony defect itself often does not require treatment, however, the associated glenohumeral instability and often co-existent anterior labral injuries often do require surgical repair.
The bony defect can be treated with bone grafting or placement of soft tissue within the defect, but this is generally reserved for large, engaging defects . Capsulotendinosis and filling of the Hill-Sachs lesion can be performed via open (Connolly procedure) or arthroscopic (remplissage) approaches .
History and etymology
It was first described in 1940 by American radiologists Harold Arthur Hill (1901-1973) and Maurice David Sachs (1909–1987) . The "engaging" Hill-Sachs was described by Burkhart and De Beer in 2000 .
Differential diagnosis
- Hatchet sign of ankylosing spondylitis
- humeral head pseudolesion
Siehe auch:
- Bankart-Läsion
- Schulterluxation
- anterior glenolabral injuries
- vordere Schulterluxation
- Musculus infraspinatus
- Hill-Sachs-Läsion im Ultraschall
und weiter:
Assoziationen und Differentialdiagnosen zu Hill-Sachs-Läsion:
