hypoxic-ischemic encephalopathy

Anoxic brain

injury following a hanging. Note the loss of grey white matter differentiation and small ventricles due to brain swelling.

Acute carbon

monoxide poisoning and hypoxic-ischaemic brain injury. Bilateral basal ganglia low density (lenticular nucleus, head of the caudate nucleus and thalamus) and diffuse hypo-attenuation of gray matter in cerebral cortex with relative preservation of white matter.

Infant who is

status post cardiac arrest. Axial CT without contrast of the brain shows decreased density of the cerebrum when compared to the density of the cerebellum. There is also loss of gray matter-white matter differentiation and effacement of the cerebral sulci and the basal cisterns. There is linear increased density present along the entire falx.The diagnosis was hypoxic ischemic encephalopathy and interhemispheric subdural hematoma in a child abuse patient.

Newborn with

failure to progress during delivery due to dystocia who is now apneic. Axial CT without contrast of the brain shows a cresenteric high-density fluid collection in the subcutaneous tissues of the right scalp that crosses suture lines and a cresenteric low-density fluid collection in the subcutaneous tissues of the left scalp that crosses suture lines. Intracranially, there is diffuse loss of gray matter-white matter differentiation secondary to diffuse cerebral edema.The diagnosis was a left caput succedaneum and a right subgaleal hematoma in a patient with hypoxic ischemic encephalopathy.

Newborn who

experienced prolonged labor. Axial (above), coronal (below left) and sagittal (below right) CT without contrast of the brain show a large low density fluid collection in the subcutaneous tissues of the scalp that crosses sutures and is seen to surround the skull on the coronal view. Intracranially, there is diffuse loss of gray matter-white matter differentiation secondary to diffuse cerebral edema.The diagnosis was caput succedaneum in a patient with hypoxic ischemic encephalopathy.

Anoxic brain

injury • Global hypoxic ischemic encephalopathy - Ganzer Fall bei Radiopaedia

White

cerebellum sign • Diffuse hypoxic ischemic brain injury - Ganzer Fall bei Radiopaedia

White

cerebellum sign • Anoxic brain injury - Ganzer Fall bei Radiopaedia

Anoxic brain

injury • Anoxic brain injury - Ganzer Fall bei Radiopaedia

Anoxic brain

injury • Anoxic brain injury - Ganzer Fall bei Radiopaedia

Acute carbon

monoxide poisoning and hypoxic-ischaemic brain injury. Diffuse hypoattenuation of gray matter in cerebral cortex with relative preservation of white matter and effacement of cerebral sulci.

Acute carbon

monoxide poisoning and hypoxic-ischaemic brain injury. Diffuse hypoattenuation of gray matter in cerebral cortex and effacement of cerebral sulci, obliteration of the fourth ventricle and the basal cisterns.

This image

is part of a series which can be scrolled interactively with the mousewheel or mouse dragging. This is done by using Template:Imagestack. The series is found in the category Profound hypoxia case 001. Computertomographie des Schädels bei einem Patienten nach generalisierter Hypoxie: Besonders die stoffwechselintensiven und somit hypoxieempfindlichen Regionen wie Globus pallidum, Hippocampus und auch die Hirnschenkel sind betroffen. Auch die Mark-Rinden-Differenzierung ist deutlich vermindert.

Hypoxic

ischaemic encephalopathy. FLAIR Hyperintensity seen in both cerebellar hemispheres.

Hypoxic

ischaemic encephalopathy. T2 hyperintensity seen in both cerebellar hemispheres.

Hypoxic

ischaemic encephalopathy. Both cerebral hemispheres, basal ganglia and thalami show low ADC map values.

Hypoxic

ischaemic encephalopathy. Diffusion restriction seen in both cerebral hemispheres, basal ganglia and thalami.

Hypoxic

ischaemic encephalopathy. Diffusion restriction seen in both cerebellar hemispheres.

Hypoxischer

Hirnschaden - Pseudo-SAB

Pseudo-SAB

bei hypoxischem Hirnschaden

Schwerer

hypoxischer Hirnschaden mit aufgehobener Mark-Rinden-Differenzierung. Computertomographie axial.

Computertomographie

des Schädels bei einem jungen Patienten nach generalisierter Hypoxie: Die stoffwechselintensiven und somit hypoxieempfindlichen Regionen hier vor allem Globus pallidum sind betroffen und somit deutlich hypodens.

Newborn with

a nuchal cord. Axial CT without contrast of the brain (above) shows diffuse cerebral edema causing loss of the gray matter white matter junction. Axial T1 without contrast (middle left), FLAIR (middle middle) and T2 (middle right) MRI several days later show developing laminar necrosis. Diffusion weighted imaging (below) shows diffusion restriction throughout the gray matter, white matter, and basal ganglia.The diagnosis was severe hypoxic ischemic encephalopathy.