anoxic brain injury

Anoxic brain

injury following a hanging. Note the loss of grey white matter differentiation and small ventricles due to brain swelling.

This image

is part of a series which can be scrolled interactively with the mousewheel or mouse dragging. This is done by using Template:Imagestack. The series is found in the category Profound hypoxia case 001. Computertomographie des Schädels bei einem Patienten nach generalisierter Hypoxie: Besonders die stoffwechselintensiven und somit hypoxieempfindlichen Regionen wie Globus pallidum, Hippocampus und auch die Hirnschenkel sind betroffen. Auch die Mark-Rinden-Differenzierung ist deutlich vermindert.

Computertomographie

des Schädels bei einem jungen Patienten nach generalisierter Hypoxie: Die stoffwechselintensiven und somit hypoxieempfindlichen Regionen hier vor allem Globus pallidum sind betroffen und somit deutlich hypodens.

Schwerer

hypoxischer Hirnschaden mit aufgehobener Mark-Rinden-Differenzierung. Computertomographie axial.

Pseudo-SAB

bei hypoxischem Hirnschaden

Hypoxischer

Hirnschaden - Pseudo-SAB

Hypoxic

ischaemic encephalopathy. Diffusion restriction seen in both cerebellar hemispheres.

Hypoxic

ischaemic encephalopathy. Diffusion restriction seen in both cerebral hemispheres, basal ganglia and thalami.

Hypoxic

ischaemic encephalopathy. Both cerebral hemispheres, basal ganglia and thalami show low ADC map values.

Hypoxic

ischaemic encephalopathy. T2 hyperintensity seen in both cerebellar hemispheres.

Hypoxic

ischaemic encephalopathy. FLAIR Hyperintensity seen in both cerebellar hemispheres.

Acute carbon

monoxide poisoning and hypoxic-ischaemic brain injury. Diffuse hypoattenuation of gray matter in cerebral cortex and effacement of cerebral sulci, obliteration of the fourth ventricle and the basal cisterns.

Acute carbon

monoxide poisoning and hypoxic-ischaemic brain injury. Bilateral basal ganglia low density (lenticular nucleus, head of the caudate nucleus and thalamus) and diffuse hypo-attenuation of gray matter in cerebral cortex with relative preservation of white matter.

Acute carbon

monoxide poisoning and hypoxic-ischaemic brain injury. Diffuse hypoattenuation of gray matter in cerebral cortex with relative preservation of white matter and effacement of cerebral sulci.